Sinus bradycardia is a condition characterized by a heart rate that falls below the conventionally set normal limit of 60 beats per minute. This condition is a type of arrhythmia, which signifies a deviation from the normal heart rhythm. However, sinus bradycardia does not involve any irregularities in the heartbeat.
The heart is a unique organ composed of a specific muscle type known as the myocardium. This muscle has the remarkable ability to autonomously generate and conduct impulses, leading to the contraction of the heart's atria and ventricles. The origin of these impulses, akin to electrical signals, is located in the heart's right atrium, specifically in the sinoatrial node.
The sinoatrial node is responsible for setting the correct contraction frequency of the heart. When the heart contracts under the influence of the sinoatrial node, it is referred to as a normal heart rhythm or sinus rhythm. The normal heart rhythm of an adult human at rest ranges between 60 and 100 beats per minute.
Sinus bradycardia is a cardiac arrhythmia characterized solely by a decrease in heart rate below the minimum limit of 60 beats per minute. This condition can have either a physiological or pathological significance.
Physiological sinus bradycardia may occur in response to consistent sports training or during sleep, when the heart rate naturally slows down. Pathological sinus bradycardia, on the other hand, results from conditions such as sinus node syndrome, hypothyroidism, hypothermia, anorexia nervosa, and others.
Sinus bradycardia can occur due to various physiological or pathological factors. Physiological causes include factors like rest during the night or an increase in vagal tone as a result of sports training. Pathological causes can range from dysfunctions of the sinoatrial node, hypothermia, hypothyroidism, anorexia nervosa, to certain infectious diseases, and certain medication use.
Physiological sinus bradycardia typically does not cause any problems. However, pathological sinus bradycardia can result in symptoms such as hypotension, dizziness, syncope, dyspnea, chest pain, exercise intolerance, peripheral edema, mild cyanosis, and reduced peripheral perfusion. It's worth noting that a significant number of pathological sinus bradycardia cases are asymptomatic, meaning they present without obvious symptoms or signs.
The cornerstone test for diagnosing sinus bradycardia is an electrocardiogram (ECG). This test reveals the rhythm and electrical activity of the heart, drawing a characteristic path on graph paper. A normal ECG has five relevant elements or waves, identified as P, Q, R, S, and T. In sinus bradycardia, the ECG would show P waves with a frequency lower than 60 beats per minute and a slower but regular pace.
The treatment for sinus bradycardia depends on its underlying causes. Physiological sinus bradycardia generally does not require any treatment. However, pathological sinus bradycardia treatment depends on the causes that triggered the arrhythmia. Common treatments include the administration of atropine and the installation of a pacemaker.
The prognosis for physiological sinus bradycardia is generally positive. However, the prognosis for pathological sinus bradycardia depends on the underlying causes. With the availability of pacemakers, prognostic predictions for pathological sinus bradycardia have significantly improved.